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Note: This site is not a Microsoft Official Site. The publisher of this site is not affiliated with Microsoft Corporation in any way, nor is this a microsoft product.Three aminobenzoate herbicides are toxic to Spirorchis, the eel-infective schistosome of the freshwater cercariae, and they target the same enzyme, ferrochelatase.
Ferrochelatase is a heme-containing enzyme required for the synthesis of heme in both the parasite, Spirorchis, and its host, the freshwater snail, Biomphalaria glabrata. This study determined the cytotoxicity of three aminobenzoate herbicides (imazapic, imazethapyr, and imazaquin) to 3 organ systems (proliferative tissue, reproductive tissues, and developing embryos). Molluscs are the intermediate host of S. mansoni. Ferrochelatase is strongly inhibited by aminobenzoate herbicides in M. littoralis; however, larval development is not significantly inhibited, suggesting that the ferrochelatase enzyme is not absolutely essential for larval development. Growth inhibition and induction of encystment occur in Biomphalaria glabrata infected with Spirorchis, and one or more metabolite(s) of the herbicide(s) is hydrolyzed in the snail. We therefore tested whether B. glabrata is also susceptible to imazapic, imazethapyr, and imazaquin. All three herbicides were equally toxic, rapidly killing B. glabrata during the first 24 hours and inhibiting worm development (female oviposition and egg maturation). The embryos of B. glabrata exposed to imazaquin were significantly shorter than those of controls, and the larvae of B. glabrata exposed to imazaquin were almost completely incapable of metamorphosis into adults. In addition, imazaquin caused an abnormal movement of the gut of treated parasites, which suggests that imazaquin is interfering with nutrition.var fs =